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Furthermore, emerging evidence suggests that melittin inhibits fungal growth via several processes, such as membrane permeability and apoptotic activation via the ROS-induced mitochondrial/caspase-dependent route, suppression of (1,3)-D-glucan synthase, and alterations in fungus genetic transcription

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The magnitude of heart rate increase in clinical trials was generally small and not clinically significant for most participants

n 25 dendritic segments)

This mechanism mirrors how minoxidil works, but with important differences

The distinction is about mechanism breadth, not potency