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One potential mechanism is the Nrf2-ARE pathway, which induces a phase 2 detoxification response that promotes disruption of Nrf2-Keap protein interactions, translocation of Nrf2 to the nucleus, and regulation of target gene expression through the ARE, enhancing cellular defense against oxidative damage (Hu et al., 2004

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This service aims to identify specific genetic markers associated with hereditary predispositions to skin barrier dysfunction and immune hypersensitivity

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